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Results Development of the secreted Exe4 peptide-coding gene To develop a one-shot in vivo genome editing-based therapy for DIO and pre-diabetes, we targeted the liver due to its large size and ease of transduction, anticipating effective therapeutic peptide/protein production and secretion following successful genome editing (Fig

(2) decreases in systolic blood pressure

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Our findings provide a new mechanistic insight into the resistance of IGF-1R inhibition in which MEK1/2 activation stimulates the survival signal (Fig