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Studies have examined its activity in musculoskeletal tissue models including tendon, ligament, and bone repair research, as well as gastrointestinal cytoprotection and neurological models

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The proposed mechanisms underlying anodal ctDCS-induced neuromodulation are derived from direct current findings in animal slices and primary motor cortex (M1) tDCS in humans [272, 273]

discomfort is minimal and temporary

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