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GIPR/GLP1R agonism along with individual GIPR agonism or GLP1R agonism reduced the expression of TNF- and proatherogenic peptides and systemic inflammation markers ICAM-1 and VCAM-1 involved in monocytes/macrophages adhesion to endothelium

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The LC treatment led to a decrease in the serum levels of tumor necrosis factor- (TNF-) and interleukin-6 (IL-6) [8, 9]

Much of the work comes from overlapping authors and institutions

FDA flagged both as potentially clinically relevant signals that warrant caution