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Results Inflammation-driven PE demonstrates evidence of NAD + depletion and mitochondrial dysfunction Previous gene set enrichment carried out by our group using a genome-wide microarray dataset from human placental samples annotated according to the PE subclass (Table 1) identified an overexpression of several pro-inflammatory signaling pathways uniquely within one PE subclasslabeled inflammation-mediated PE (PE3) (3, 4)

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The bulk of litigation dealt with Crohns disease and ulcerative colitis

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