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Dysregulated complement activation during acute myocardial infarction leads to endothelial glycocalyx degradation and endothelial dysfunction via the C5a: C5a-receptor 1 axis

Ahmadi M, Rajaei Z, Hadjzadeh M, Nemati H, Hosseini M

Therefore, persistent interactions with misfolded or metastable proteins can overload the chaperone system, thereby ultimately compromising global protein homeostasis

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