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Despite the consensus on the role of genetic predisposition and the greater susceptibility of vitiligo development, in monozygotic twins, vitiligo develops in only 23% of both twins (Alkhateeb et al., 2003), suggesting that the environment can induce or suppress the genes related to vitiligo development via DNA methylation, histone modifications and alteration of circulating microRNA (Zhou et al., 2019)

Specifically, liposomal MVM ingestion resulted in lower volume distribution of vitamins compared to non-liposomal MVM sources, suggesting improved clearance and absorption despite similar nutrient intake

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