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Characterizing the mechanism of action for mRNA therapeutics for the treatment of propionic acidemia, methylmalonic acidemia, and phenylketonuria

Inhibition of Krebs cycle in normal prostate cells leading to accumulation and secretion of citrate, which is predominantly obtained via the suppression of m-aconitase, due to an accumulation of zinc in normal prostate cells [85]

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if too much medication is taken, these stores are used up, potentially leading to oxidative stress in the liver