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HGF/c-Met signaling is a well-established oncogenic pathway: c-Met is amplified or overexpressed in multiple cancers (gastric, lung, renal, hepatocellular) HGF/c-Met activation promotes tumor cell proliferation, survival, invasion, and metastasis c-Met inhibitors are FDA-approved cancer drugs (cabozantinib, crizotinib, capmatinib) A compound that potentiates HGF/c-Met signaling raises theoretical concerns about promoting tumor growth or transformation with chronic use

To view a copy of this licence, visit About this article Cite this article Kong, YX., Li, ZS., Liu, YB

[2] Is GHK-Cu the same as a copper peptide serum

Longitudinal human work is scarce

Gradual dose titration and practical dietary strategies can help minimise these effects during the initial period