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In humans, administration of 2 g/day of ALCAR for 50 days increased plasma ALCAR concentrations by 43%, suggesting that either ALCAR was absorbed without prior hydrolysis or that L-carnitine was re-acetylated in the enterocytes (5)

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In these renal failure patients, not only reduced free carnitine, but also accumulation of acylcarnitine is observed resulting in carnitine insufficiency (high acylcarnitine concentration/free carnitine concentration (AC/FC)) which inhibits mitochondrial functions