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The proposed mechanisms underlying these protective effects include the inhibition of several signaling pathways implicated in cancer initiation and progression, such as the phosphoinositide 3 kinase (PI3K)/protein kinase B (AKT)/mammalian target of rapamycin (mTOR), extracellular signal-regulated kinase/microtubule-associated protein kinase, and nuclear factor kappa-light-chain-enhancer of activated B cell pathways [32-34]

Mechanisms of AMPK in the maintenance of ATP balance during energy metabolism

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FFAs induce oxidative stress by generating ROS, which promote lipid peroxidation and activate cell death pathways, thereby exacerbating the vicious cycle between mitochondrial failure and ER stress (Khoi et al., 2024