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What is the future of GLP-1 receptor agonists

Simultaneously, skeletal muscle in the elderly demonstrates mitochondrial dysfunction, encompassing diminished mitochondrial biogenesis, compromised oxidative phosphorylation, and an augmented production of reactive oxygen species (ROS), which further intensify endothelial oxidative damage and inflammatory responses (21, 22)

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On the other hand, the GSH precursor induces an increase in mitogen-stimulated cellular proliferation in HTLV-1-infected individuals [19]