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An example is the influence of the gut on neurotransmitters and vice versa is the abovementioned monoaminergic system [76, 87, 232], where reduced DA and adrenaline in the gut, which led to a decrease in anxiety-like behavior, indicating probably a link of these neurotransmitters with behavior

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Numerous reports demonstrate significant impairments in intestinal motility (small and large) in TD subjects, which normalizes with thiamine repletion (18, 7578)

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It increases growth hormone receptor expression in tendon fibroblasts at both mRNA and protein levels, with effects lasting at least three days