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Modulation of inflammatory pathways and reduction in systemic inflammation

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Future research should continue to explore the impact of commensal translocation on AD development/progression to facilitate the development of therapeutics that perhaps target the microbiota to indirectly inhibit chronic inflammation in the gut and beyond

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Similar content being viewed by others Background The mesenchymal-epithelial transition (MET) can be utilized as a signaling receptor for hepatocyte growth factor (HGF), and it has been reported that in humans, the MET proto-oncogene encodes for the receptor tyrosine kinase (RTK) of growth factor tyrosine kinase [1, 2]