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In the central nervous system, GLP-1 RAs can either directly cross regions with a compromised blood-brain barrier or act via vagal afferent signaling to target GLP-1 receptors (GLP-1Rs) in the nucleus tractus solitarius (NTS) of the brainstem

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Even though current therapies are effective for improvement of HF symptoms and provide an acceptable clinical benefit, the marginal impact on HF outcome has still emerged such as an increase in rate of mortality and hospitalization (Yancy et al., 2013)

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