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PIP markedly reduced CS-induced (crystalline silica) inflammation and fibrosis in vivo by suppressing inflammatory cell infiltration, inflammasome formation, collagen/ECM deposition, and inflammatory and fibrotic mediators

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In immature osteoblasts in vitro, GCs exposure was associated with cell cycle arrest in G1 phase due to down-regulation of cell cycle activators such as cyclin A, cyclin D, cyclin-dependent kinase 2 (CDK2), CDK4 and CDK6 [21,22,23], as well as activation of cell cycle inhibitors such as p53, p21 and p27 [21, 24]

372 Ferroptosis also occurs in peripheral blood monocytes in SLE, with electron microscopy revealing characteristic ferroptosis features, such as decreased mitochondrial volume, increased mitochondrial membrane density and the disappearance of mitochondria

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