and Tamar Weiss Prize Date: May 1, 1993 Event: Location: Dickinson College David M Glasgow Emil R
Its believed to promote musculoskeletal repair by upregulating growth factors, stimulating the formation of new blood vessels, or modulating nitric oxide synthesis, but no mechanism of action has been proven.34 TB4, however, has known mechanisms by which it speeds recovery from injury: inhibiting inflammation, preventing cell death, and (as suspected with BPC) promoting blood vessel formation.35 On top of the benefits for physical recovery, the ability to form new blood vessels points to promising implications for cardiovascular health
In DECLARE-TIMI58 with dapagliflozin, 4.4% of the patients were using GLP-1RAs at baseline, and dapagliflozin reduced the risks of MACE, HHF, and composite kidney outcomes regardless of baseline GLP-1RA use [49]
Although not currently prescribed for human use, it is important for athletes to proceed with caution when looking at potential agents to prevent and treat injury
Raghu G, Berk M, Campochiaro PA, Jaeschke H, Marenzi G, Richeldi L et al
