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To view a copy of this licence, visit About this article Cite this article La, P.L., Walker, R., Bell, T.K

Similarly, NSC34 motor neuron-like cells stably expressing the human G93A mutant form of SOD1 displayed a significant and selective depletion of mitochondrial GSH content in comparison to parental cells, reminiscent of some forms of familial ALS [16]

(26) The greatest risk reduction occurred in long-term users (HR: 0.44, 95% CI: 0.270.72)

20 This observation emphasizes their potential to promote systemic metabolic health beyond glucose homeostasis and makes GLP-1RAs a more suitable treatment option for T2D patients with a higher risk of cardiovascular disease compared to metformin

Neuropharmacology (2021) 195:108662