By inhibiting molecular pathways driving fibroblast activation and collagen dysregulation, anti-fibrotic therapies such as pirfenidone and nintedanib demonstrate clinical potential to attenuate radiation-triggered tissue stiffening while preserving alveolar-capillary gas exchange capacity [27, 28]
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Because half-life varies from a few minutes to several days across different peptides, accurate reference data from a reliable peptide half-life chart is essential for experimental design
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